Atherosclerosis interactive video

If the foam cells are unable to process the oxidized ldl particles and recruit high-density lipoprotein (HDL) particles to remove the fats and cholesterol, then foam cells will continue to expand and eventually rupture. Rupture of foam cells results in the release of oxidized materials and fats in the artery wall that causes yet another cycle of inflammatory responses that attract more white blood cells and further inflames the artery. Fatty streaks will therefore contain both active cells that produce inflammation in addition to dead cell remnants. At this point, there is a migration of vascular smooth muscle cells (vsmcs) to these sites of inflammation, where endogenously released substances including platelet-derived growth factor (pdgf), fibroblast growth factor (fgf endothelin-1, and il-1 influence additional vsmc migration and proliferation. Recent research has shown that vsmcs respond to vascular injury by switching from the quiescent contractile phenotype to a proinflammatory phenotype. Additionally, there will be deposition of extracellular matrix by vsmcs and further T-cell recruitment to these sites.

atherosclerosis interactive video
Vitamins and progression of atherosclerosis in hyper-homocyst(e

Vldl molecules are produced by the liver from triacylglycerol and cholesterol from the diet that was not used during the synthesis of bile acids. These vldl molecules then lose triglyceride through the action of lipoprotein lipase, thus making them smaller and denser, containing a higher proportion of cholesterol esters. Lowered protein/lipid ratios result in less dense lipoproteins. Ldl particles voor function as the primary major cholesterol carriers in the blood, with each particle containing approximately 1,500 molecules of cholesterol ester. It is thought that the retention of ldl particles, in conjunction with inflammation, is what results in endothelial cell dysfunction that instigates the process of atherosclerosis. Ldl particles and their content are susceptible to oxidation by free radicals, and are more prone to oxidation once inside the blood vessel wall. As a result, endothelial cells respond to these inflammatory processes by attracting monocyte white blood cells from the blood into the arterial walls, therefore transforming them to macrophages. Endothelial cells Therefore following endothelial cell dysfunction, there is an accumulation of white blood cells, as mostly macrophages and ldl particles. Macrophages (and some t-lymphocytes) ingest oxidized ldl particles, which results in foam cell formation, otherwise described in histopathology as lipid-laden macrophages. Buildup of these lipid-laden macrophages appear grossly as a thin layer of yellowish-white fatty streaks within the inner layers of artery walls beginning during childhood, and continue expanding over time.

atherosclerosis interactive video
Atherosclerosis - cardiovascular Disorders - merck manuals

Atherosclerosis - cardiovascular Disorders - msd manual


Ascending aorta - example of a hpv large elastic artery medium-sized muscular arteries (e.g., coronary and renal arteries) tijdens - the media is composed primarily of smooth muscle cells, with elastin limited to the internal and external elastic lamina. Left coronary artery - example of a medium-sized muscular artery Small arteries (2 mm or less in diameter) and arterioles (20 to 100 μm in diameter) - the media in these vessels is mostly composed of smooth muscle cells. Blood flow resistance is regulated at the arterioles and therefore pressure will drop during passage through the arterioles, causing blood flow to become more steady rather than pulsatile. This is due to the fact that under poiseuilles law, the resistance to fluid flow is inversely proportional to the fourth power of the diameter (i.e., halving the diameter increases resistance 16-fold which means that small changes in arteriolar lumen size will have profound effects. Pathogenesis Inflammatory processes that set off a cascade of responses and changes over time initiate atherosclerosis. However, while a degree of atherosclerosis is found in a large majority of the population, its exact pathogenesis is still under debate. Typically, atherosclerosis begins in childhood with endothelial cells of the vessel wall, thought to be those associated with retained low-density lipoprotein (LDL) particles. Liver is the organ where the vldl molecules are produced ldl particles are formed initially as vldl lipoproteins.


Pima Indians hhmi biointeractive


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atherosclerosis interactive video
Aha media player - watchlearnlive

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055 Regulating Peripheral Resistance - part

Keynote lecture by Brian. Smedley, phD, will be webcast, with live viewer questions, ( info ) Social Determinants of health and Disparities: moving the nation to care about Social Justice 18th National health Equity research Webcast (formerly the Annual Summer Public health Research Videoconference on Minority health), recorded June. 24, 2012 ( info ). Archived webcasts available from the 14th Annual William. Keynote lecture by Ana diez-roux and the 1st operatie Annual Victor. Schoenbach health Disparities keynote lecture by nina wallerstein. ( webcasts ) health Equity: Progress and Pitfalls 17th Annual Summer Public health Research Videoconference on Minority health, june 7, 2011 ( info ) The Promise of health Equity: Advancing the discussion to Eliminate disparities in the 21st Century 32nd Annual Minority health Conference, with. Keynote lecture by bonnie duran.

atherosclerosis interactive video
The Science of Fat hhmi biointeractive

What is Cardiovascular Disease?

Keynote lecture, february 23, 2018 ( info, video announcement the courage to lead: Scholar-Activism and health biologische Equity in Turbulent Times 23rd National health Equity research Webcast, recorded September 29, 2017, ( webcast systems of Power: Recalling Our Past, restructuring Our Future 38th annual Minority health. Keynote lecture, february 24, 2017 ( info, video announcement ) Political Power, policy, and health Equity 22nd National health Equity research Webcast, recorded June 7, 2016, ( webcast ) Recorded presentations of my current thinking, including: Why havent compelling epidemiologic data eliminated health disparities?, first. Keynote lecture, february 26, 2016 ( info, video announcement ) Advancing a community- based Model for violence Prevention 21st National health Equity research Webcast, recorded June 2, 2015, ( webcast ) reaching for the American Dream: Economic Mobility and Minority health 36th annual Minority health. Keynote lecture, presented by william. Darity, phd, february 27, 2015 ( info, video announcement ) School-to-Prison Pipeline: from Perceptions to solutions 20th National health Equity research Webcast, recorded June 3, 2014, ( webcast ) Innovative approaches to youth health: Engaging youth in Creating healthy communities 35th annual Minority health Conference. Keynote lecture, presented. Gail Christopher, february 28, 2014 ( info, video announcement ) Early Childhood development: Investing in Our Children and Our Future 19th National health Equity research Webcast (formerly the Annual Summer Public health Research Videoconference on Minority health), recorded June 4, 2013 ( info ) Constructing. The 15th Annual William.


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american heart Association

The tunica intima is the innermost and thinnest layer, consisting of a monolayer of simple squamous endothelial cells on top of a basement membrane with minimal underlying extracellular matrix (ECM). The intima is separated from the next layer by a dense elastic membrane called the internal elastic lamina, which are circularly arranged bands. Tunica media of artery, the tunica media is the thickest layer of the arteries, and is composed predominantly of smooth muscle cells and ecm, surrounded by loose connective tissue, nerve fibers, and smaller vessels of the adventitia. Separating the media from the adventitia tadalafil is another elastic band known as the external elastic lamina. Tunica Adventitia of artery The tunica adventitia is the thickest layer in the veins, and is entirely composed of connective tissue. However, in large and medium-sized vessels, the adventitia contains nerves and small arterioles (nutrient capillaries called the " vasa vasorum literally, "vessels of the vessels that supply that vessel and the outer half to two thirds of the media. Additionally, arteries are divided into three types based on their size and structure: Large elastic arteries (e.g., aorta, arch vessels, iliac and pulmonary arteries ) - in this type, elastic fibers alternate with smooth muscle cells throughout the media, which expands during systole (storing some. This elasticity is increasingly lost with age.


a result of invasion and accumulation of white blood cells and proliferation of intimal smooth muscle cells that create a fibrofatty plaque. Atherosclerosis is thus a syndrome affecting arterial blood vessels due to a chronic inflammatory response in the artery walls. While atherosclerosis often remains asymptomatic for decades, it can increase the risk of local thrombosis and distal embolization that can present clinically as myocardial infarction or stroke. Inner circular smooth muscle layer - histological slide. Recommended video: Coronary arteries and cardiac veins. Overview of the main coronary arteries and cardiac veins. Anatomy of Arteries, artery - histological slide, vessel walls of arteries and veins are organized into 3 concentric, similar layers : the tunica intima, the media and, the adventitia. However, the middle layer (tunica media) is thicker in arteries than in veins. Tunica intima of artery.
Atherosclerosis interactive video
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Recensies voor het bericht atherosclerosis interactive video

  1. Wyxoric hij schrijft:

    Below is an approximation of this video s audio content. To see any graphs, charts, graphics, images, and"s to which. Greger may be referring, watch the above video. Or lower extremities is the narrowing or blockage of the vessels that carry blood from the heart to the legs.

  2. Axupubo hij schrijft:

    Atherosclerosis is a condition that develops when a substance called plaque builds up in the walls of the arteries. This buildup narrows the. Cardiology : Welcome to theheart. Org medscape cardiology, where you can peruse the latest medical news, commentary from clinician experts, major conference coverage, full-text journal articles, and trending stories.

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    Evans and Jeffrey. Friedman discuss how the body regulates weight by carefully controlling the storage and burning of fat—and how a better understanding of these complex metabolic systems could lead researchers to treatments that curb obesity and improve public health. Heart and blood vessel disease — also called heart disease — includes numerous problems, many of which are related to a process called atherosclerosis.

  4. Togunap hij schrijft:

    Materials for patients and health professionals on health topics related to overweight and obesity, heart, lung, blood, and sleep disorders. As promised, here is a video focusing on peripheral resistance. Understand what it is and how it affects mean arterial pressure by watching along and listening as Leslie once again explains the concept with full clarity. In the 2004 Holiday lectures on Science, hhmi investigators Ronald.

  5. Ysyci hij schrijft:

    A clip about the pima Indian tribe and how environment has affected them. Watch, learn and live with the American heart Association s interactive cardiovascular library. Here you ll find informative illustrations and animations about a variety of conditions, treatments and procedures related to heart disease and stroke.



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